How does normal function return after experiencing pulmonary O2 toxicity?

Normal function returns after experiencing pulmonary oxygen toxicity primarily when exposure to high levels of oxygen is terminated and the supply is removed. Inhalation of oxygen at high pressures or concentrations can lead to damaging effects on the lung tissues, primarily due to the formation of reactive oxygen species, which can result in inflammation and edema. Once the individual is removed from the hyperoxic environment, the lungs begin to recover, allowing for the resolution of these toxic effects.

The natural physiological response includes repair mechanisms that the body initiates upon return to normal oxygen levels. This healing process may involve the restoration of normal cell function and lung homeostasis.

Other options may suggest alternative healing processes, but they do not accurately capture the primary mechanism through which recovery occurs in this context. The correct approach emphasizes the importance of removing the source of oxygen toxicity as the critical step for resolution and return to normal lung function.